Post Vaccination - Vaccine Targeted Strain - Viral and Bacterial Pathogen - Shedding
So how much of this said claim is truth and real, and/or not real? Do we know? A search for the evidence.
First of all, let me ask this. Why is it, that when the conclusions of actually peer reviewed studies are not in your favor as to the intended agenda bias, that even endless peer reviewed studies are not enough to get pro-vaccine people to take a look at and even read a single one of those studies; yet when there are limited to little to no existing peer reviewed studies, that they are jumping all over with demands to produce a peer reviewed study, to make such as any certain such as a vax-truth opposing persons point of contention, that has expressed?
In regard to vaccines lets go to the issue of vaccine shedding, and ask the question as to can and do any of the current vaccines shed the pathogen in a way that could make a non vaccinated person susceptible to acquiring the illness from a vaccinated person. There are in Pubmed several but limited studies that address the shedding issue as to in regard to the various vaccines. Just use the search terms vaccine shedding Pubmed, and will you several but as said limited numbers references in the google listing, and then you can go to pubmed itself, which is somewhat as well limited for available references as to claiming one way or the other. As for the measles vaccine, one Pubmed reference stated that it the vaccine could shed for up to three days. Certainly long enough to infect another individual.
So, actually and possibly no one really nor likely knows for sure what the complete truth is on this issue. It would seem to be common sense that the vaccine makers surely do not and would not want to know if their vaccine causes shedding or not; nor to find out. So then who would actually fund theses said studies. I think with what I read and reviewed in regard to vaccine shedding, just getting into even the beginning phase of the studies, tells me that vaccines do have a potential to shed irregardless of being bacterial or viral; which very well could be an obvious risk to the unvaccinated. I mean good grief, the existing studies clearly point to the push to vaccinate everyone due to the risk of shedding possibility. What more evidence would you need of the risk of the vaccinated, to the unvaccinated? And yet the pro-vaccine side wants to claim to just the opposite; and that it is only the vaccinated that are at risk from the unvaccinated??? You know accused again of reducing the vaccine derived herd immunity; even though the schools most often even today have no more than a 5% or less rate of existing school exemptions? We as well by the way are not are NOT just talking about the oral polio vaccine, here. They clearly know that the oral polio vaccine sheds and can as well cause numerous cases of AFP in the underdeveloped and unsanitary for conditions countries, that the oral polio vaccine is still used to day. They know of the identified mutations in the polio vaccine virus that the the said oral vaccine has very likely as well caused. if they have an alternative explanation, I have yet to hear and or read about it.
So, let me ask you, have vaccines eradicated so called illness and disease, or have they just prolonged the exit, while creating only lower levels of chronic disease, and disease conditions? How about other unrelated chronic illness and autoimmune disease, unrelated to the vaccine targeted pathogen? How about the pubmed listed as well references to the harm of aluminum adjuvants, causing overactivation of the brains microglia and resulting low levels of chronic brain inflammation resulting for repeat multiple vaccines, in some individuals; maybe more individuals and children that we have ever realized? How about the aluminum adjuvant connection to ASD? The studies, and new studies have shown that same brain inflammation to now be found in more and more children and individuals with ASD. And they want to tell us that vaccines have never been scientifically linked in any study, to ASD? Really? How about the MMR vaccine, in which there are actually some similar physiological pathways found in relation to ASD, and also which are in common with heavy metal toxicity, if not overload, in regard to both thimerosal, and aluminum adjuvants. I don't know about you and what you think, but I think it is not looking good for the claim as to the issue of vaccines doing more good than harm. When will the CD stop living in the dark ages, and dragging their feet as to doing the proper studies? Yet they waste millions chasing the genetic link to ASD, and refuse all other types and forms of real research?
I did pick one specific peer reviewed reference in regard shedding, that I thought was interesting, and a bit troublesome regarding risk. In regard to the shedding of course all they can come up with is to come up with that every last person existing must be vaccinated to protect them against the shedding.
Pertussis infection in fully vaccinated children in day-care centers, Israel.
We tested 46 fully vaccinated children in two day-care centers in Israel who were exposed to a fatal case of pertussis infection. Only two of five children who tested positive for Bordetella pertussis met the World Health Organization's case definition for pertussis. Vaccinated children may be asymptomatic reservoirs for infection.
And they tell us the vaccines do not shed? How would this be possible if the vaccines do not shed anything contagious? And they want us to believe that the un-vaccinated are a risk to the vaccinated. Vaccine derived herd relatively and comparatively short term immunity, has never had any actual science behind it; and as to natural long term and/or life time immunity, where as that concept actually makes does sense. So what is the REAL reason they say they need vaccine derived herd immunity? Is it possible that it is more likely due to the issue of vaccine shedding? Now we are getting to some actual understanding of what possibly really goes on.
In fact this below study shows that vaccinating infants at two months of age and potentially even older, does again potentially little to nothing to protect infants that are to young to vaccinate. More harm than good.
J Infect Dis. 2013 May 8. [Epub ahead of print] Waning of Maternal Antibodies Against Measles, Mumps, Rubella, and Varicella in Communities With Contrasting Vaccination Coverage.
Waaijenborg S, Hahné SJ, Mollema L, Smits GP, Berbers GA, van der Klis FR, de Melker HE, Wallinga J.
Source: Department of Epidemiology and Surveillance.
Background. The combined measles, mumps, and rubella (MMR) vaccine has been successfully administered for >20 years. Because of this, protection by maternal antibodies in infants born to vaccinated mothers might be negatively affected.Methods. A large cross-sectional serologic survey was conducted in the Netherlands during 2006-2007. We compared the kinetics of antibody concentrations in children and women of childbearing age in the highly vaccinated general population with those in orthodox Protestant communities that were exposed to outbreaks.Results. The estimated duration of protection by maternal antibodies among infants in the general population, most of whom were born to vaccinated mothers, was short: 3.3 months for measles, 2.7 months for mumps, 3.9 months for rubella, and 3.4 months for varicella. The duration of protection against measles was 2 months longer for infants born in the orthodox communities, most of whom had unvaccinated mothers. For rubella, mothers in the orthodox communities had higher concentrations of antibodies as compared to the general population.Conclusions. Children of mothers vaccinated against measles and, possibly, rubella have lower concentrations of maternal antibodies and lose protection by maternal antibodies at an earlier age than children of mothers in communities that oppose vaccination. This increases the risk of disease transmission in highly vaccinated populations.
Here is the study, showing what is likely to really be going on with the pertussis DTaP, and Tdap vaccines. And do you know how much the CDC has falsely fear mongered that it is the un-vaccinated that have caused any and all outbreaks, even though with mandatory vaccination in the schools, that would be nearly impossible, as to the already met required percentage of vaccine use to acquire what they say is the necessary percentage to attain so called vaccine derived herd immunity. Then the CDC panicked and started vaccinating whole families in what they called cocoon style vaccinating, and that clearly has as well failed. So they then have done what; pushed the tetanus, diphtheria, pertussis vaccine on adults, as to their regular clinic visits, and is as well now right on the CDC recommended vaccines list for adults. No vaccine can be admitted to have failed, in the eyes of the CDC, nor be to dangerous, such as Gardasil. Either they simply do not get it because they do not want to, or the are knowingly lying and covering this up, which is it? The public spotlight is as well shining far to brightly as to concerns over vaccine safety and efficacy, and they at the CDC obviously must hold out with the cover-ups and misinformation until there is nothing left to hold onto.
Acellular pertussis vaccination enhances B. parapertussis colonization
An acellular whooping cough vaccine actually enhances the colonization of Bordetella parapertussis in mice; pointing towards a rise in B. parapertussis incidence resulting from acellular vaccination, which may have contributed to the observed increase in whooping cough over the last decade.
Despite over 50 years of population-wide vaccination, whooping cough incidence is on the rise. Although Bordetella pertussis is considered the main causative agent of whooping cough in humans, Bordetella parapertussis infections are not uncommon. The widely used acellular whooping cough vaccines (aP) are comprised solely of B. pertussis antigens that hold little or no efficacy against B. parapertussis. Here, we ask how aP vaccination affects competitive interactions between Bordetella species within co-infected rodent hosts and thus the aP-driven strength and direction of in-host selection. We show that aP vaccination helped clear B. pertussis but resulted in an approximately 40-fold increase in B. parapertussis lung colony-forming units (CFUs). Such vaccine-mediated facilitation of B. parapertussis did not arise as a result of competitive release; B. parapertussis CFUs were higher in aP-relative to sham-vaccinated hosts regardless of whether infections were single or mixed. Further, we show that aP vaccination impedes host immunity against B. parapertussis-measured as reduced lung inflammatory and neutrophil responses. Thus, we conclude that aP vaccination interferes with the optimal clearance of B. parapertussis and enhances the performance of this pathogen. Our data raise the possibility that widespread aP vaccination can create hosts more susceptible to B. parapertussis infection.
And rarely are they testing for it nor even knowing understanding what pertussis pathogen strains are there. B parapertussis antigen is not in the current vaccine. And the fear mongering and the recommended boosters continue.
They can admit to the pertussis vaccine failure in Pakistan,
but the CDC can not and refuses to admit to that here happening in the US.
Public Health. 2012 Jun;126(6):518-22. doi: 10.1016/j.puhe.2012.02.001. Epub 2012 Mar 23.
Pertussis resurgence among vaccinated children in Khairpur, Sindh, Pakistan.
Mughal A, Kazi YF, Bukhari HA, Ali M.
Source:Diagnostic and Research Centre, Department of Microbiology, Shah Abdul Latif University, Khairpur, Sindh, Pakistan.
To investigate the aetiology of persistent cough among vaccinated children as suspected cases of pertussis in Khairpur District, Sindh, Pakistan. Pertussis or whooping cough, caused by Bordetella pertussis, is re-appearing in many countries despite vaccination coverage. In Khairpur, persistent cough and symptoms similar to pertussis among vaccinated children are common but the aetiology has not been investigated previously.
B. pertussis was isolated from cough samples of suspected pertussis patients (n = 700) using the cough plate method with charcoal agar.
Isolation and confirmation of the clinical isolates of B. pertussis was performed by culture on Bordet-Gengou medium, biochemical tests and polymerase chain reaction.
In total, 22 strains of B. pertussis were isolated from clinical cough samples.
To the authors' knowledge, this is the first report of the presence of pertussis in vaccinated children in Khairpur. There is a need for continuous monitoring of pertussis after immunization programmes in order to assess the efficacy of pertussis vaccination.
And what has the CDC done about it all? They have only continued with their fear mongering and falsely blaming the un-vaccinated. Cocoon style vaccinating whole families, and still the outbreaks occur.
Bordetella pertussis Strains with Increased Toxin Production Associated with Pertussis Resurgence (PDF)
We present evidence that in the Netherlands the dramatic increase in pertussis is temporally associated with the emergence of Bordetella pertussis strains carrying a novel allele for the pertussis toxin promoter, which confers
increased pertussis toxin (Ptx) production. Epidemiologic data suggest that these strains are more virulent in humans. We discuss changes in the ecology of B. pertussis that may have driven this adaptation. Our results underline the importance of Ptx in transmission, suggest that vaccination may select for increased virulence, and indicate ways to control
Prevalent serotypes of Bordetella pertussis in non-vaccinated communities.
In many countries, the prevalent serotypes of Bordetella pertussis have changed from a mixture of types 1,2,3 and 1,2 (organisms possessing antigen 2) to a predominance of type 1,3. The timing of the change in different countries is shown to be related to the introduction of mass-vaccination with material rich in antigens 1 and 2 but weak in, or devoid of, antigen 3. In several parts of the world, there have been outbreaks of type 1,3 infection in fully vaccinated children. Non-vaccinated communities in various parts of the world still show the pattern of serotypes which existed elsewhere before mass-vaccination. In order to avoid the disappointments experienced in the past, it is essential that pertussis vaccine for use in previously non-vaccinated communities, like that for any other country, should be rich in each of the three antigens.
Small Mutations in Bordetella pertussis Are Associated with Selective Sweeps
Our results suggest that the B. pertussis gene repertoire is already well adapted to its current niche and required only fine tuning to persist in the face of vaccination. Further, this work shows that small mutations, even single SNPs, can drive large changes in the populations of bacterial pathogens within a time span of six to 19 years.
Here is what they already knew years ago in the treatment of pertussis.
Can Med Assoc J. 1937 August; 37(2): 134–136. PMCID: PMC1562195 Ascorbic Acid (Vitamin C) Treatment of Whooping Cough *
Discussion The short series of cases presented is too small to draw any statistical conclusions, but one fact stands out. Ascorbic acid has a definite efTect in shortening the period of paroxysms from a matter of weeks to a matter of days. We have not checked by cough plates or otherwise in this preliminary work to see whether the infectivity subsides simultaneously with the spasmodic symptoms, but are continuing with a larger series of cases in which these and other tests will be employed.
The dosages used have been empirical, with a tendency to use larger doses early in the disease as our experience of its effects progressed. The acid is available at reasonable prices, and the danger of overdosage seems negligible. Animals have received 2,000 times their estimated requirements without any deleterious effects. Any excess is excreted by the kidneys.
CONCLUSIONS 1. A method has been described for the treatment of whooping cough by ascorbic acid (vitamin C). 2. Ascorbic acid definitely shortens the paroxysmal stage of the disease, particularly if relatively
Nine cases of eczema vaccinatum are presented, including two fatalities. Seven were caused by contact of a child with eczema with a recently vaccinated sibling.
Suddenly appearing umbilicated vesicles superimposed upon atopic eczema are almost diagnostic of eczema vaccinatum or eczema herpeticum. These do not occur with mere secondary bacterial infection.
Hyperimmune vaccinal gamma-globulin is now available for specific therapy.
Eczema vaccinatum is frequently iatrogenic and uniformly preventable.
The following steps are recommended for prophylaxis: 1) No child with atopic eczema or other skin disorder should be vaccinated. 2) No child should be vaccinated if any member of his family has eczema or other skin disorder. 3) Parents of children with eczema should be notified at the onset of the disease of the danger from vaccination contact. 4) If a sibling of a child with atopic eczema is vaccinated, he must be completely separated from that child for at least 21 days. 5) Forms used by state and local health departments for parents' consent to vaccination should include an appropriate warning of the contraindications. 6) Eczema vaccinatum should be a reportable disease. 7) Patients recently vaccinated must be excluded from pediatric wards containing patients with atopic eczema, other diseases of the skin, burns or healing surgical incisions. 8) Vaccination may be recommended at 2 months of age, especially for babies from strongly allergic families.
Another below is another example of a failed effort with polio vaccine. It does little good to claim to have eliminated a certain number of previously present cases of polio, while at the same time causing massive cases of polio vaccine derived paralysis. 47,500 new cases. Yet they claim this is NECESSARY, to eradicate polio. They refuse to admit any failure, it seems to me?
Indian J Med Ethics. 2012 Apr-Jun;9(2):114-7. Polio programme: let us declare victory and move on.
Vashisht N, Puliyel J.
Source:Department of Paediatrics, St Stephens Hospital, Delhi 110054, India.
It was hoped that following polio eradication, immunisation could be stopped. However the synthesis of polio virus in 2002, made eradication impossible. It is argued that getting poor countries to expend their scarce resources on an impossible dream over the last 10 years was unethical. Furthermore, while India has been polio-free for a year, there has been a huge increase in non-polio acute flaccid paralysis (NPAFP). In 2011, there were an extra 47,500 new cases of NPAFP. Clinically indistinguishable from polio paralysis but twice as deadly, the incidence of NPAFP was directly proportional to doses of oral polio received. Though this data was collected within the polio surveillance system, it was not investigated. The principle of primum-non-nocere was violated. The authors suggest that the huge bill of US$ 8 billion spent on the programme, is a small sum to pay if the world learns to be wary of such vertical programmes in the future.
Experts have long believed epidemics unleashed by a vaccine's mutated virus wouldn't last since the vaccine only contains a weakened virus strain - but that assumption is coming under pressure. Some experts now say that once viruses from vaccines start circulating they can become just as dangerous as wild viruses.
"The only difference is that this virus was originally in a vaccine vial," said Olen Kew, a virologist at the U.S. Centers for Disease Control and Prevention.
The oral polio vaccine used in Nigeria and elsewhere contains a mild version of the live virus. Children who have been vaccinated pass the virus into the water supply through urine or feces. Other children who then play in or drink that water pick up the vaccine's virus, which gives them some protection against polio.
But in rare instances, as the virus passes through unimmunized children, it can mutate into a strain dangerous enough to ignite new outbreaks, particularly if immunization rates in the rest of the population are low.
Kew said genetic analysis proves mutated viruses from the vaccine have caused at least seven separate outbreaks in Nigeria.
Though Nigeria's coverage rates have improved, up to 15 percent of children in the north still haven't been vaccinated against polio. To eradicate the disease, officials need to reach about 95 percent of the population.
Nigeria's vaccine-linked outbreak underlines the need to stop using the oral polio vaccine as soon as possible, since it can create the very epidemics it was designed to stop, experts say. WHO is researching other vaccines that might work better, but none is on the horizon.
Until a better vaccine is ready, WHO and U.S. CDC officials say the oral vaccine is the best available tool to eradicate polio and that when inoculation rates are nearly 100 percent it works fine.
"Nigeria is almost a case study in what happens when you don't follow the recommendations," Kew said.
A mutation from a live polio vaccine is stalking Nigeria. In a strange twist of logic, experts are claiming that it mutated as it passed through non-immunized children.
The claim is that children given the live attenuated oral vaccine are properly immunized, but the live virus passes through them and enters local water supplies through their urine or feces. Then, children who have not been immunized pick up the supposedly safe virus by drinking or playing in the water. The weakened virus mutates in them, becoming a new virulent strain.
Why the virus would choose to mutate in non-vaccinated, rather than vaccinated, children is unexplained. Even odder is why the weakened virus would pass through the vaccinated children. If the purpose of a live attenuated vaccination is to force the body to develop antibodies to the virus, then why would live viruses be excreted? Shouldn't they be killed by the newly-developed antibodies?
Are we being lied to?
This sounds much like the argument that blames nonvaccinated people for disease in those who've submitted to innoculations. If the vaccines are effective, then why would the vaccinated be at risk from the unvaccinated?
Point mutations involved in the attenuation/neurovirulence alternation in type 1 and 2 oral polio vaccine strains detected by site-specific polymerase chain reaction.
We screened for this mutation in five type 1 and nine type 2 polio vaccine-derived strains isolated from vaccine-associated paralytic poliomyelitis (VAPP) cases and in 16 such strains isolated from healthy vaccinees. All 14 strains isolated from VAPP presented the reversion. Of the eight pairs of type 1 isolates from healthy vaccinees, four presented the reversion 3 days after vaccine administration and all but one at 7 days postvaccination. These results support the involvement of the 5' non-coding specific nucleotide sites in the reversion to neurovirulence of attenuated polio vaccine strains upon multiplication in the human gut
Look at the unbelievable statements in the next set of information. So ask, WHY are they using a live and shedding viral vaccine, in these contaminated areas, at all?
Oral Polio Vaccine Circulation and Mutation after Mexican National Immunization Weeks
Conclusion: OPV, primarily serotype 2, was detected in sewage as late as 7 months after an NIW in a Mexican community primarily vaccinated with IPV, but was not detected at 8 months, suggesting that OPV circulation may have ceased. VAPP mutants were predominantly detected. This data suggests that in communities with high vaccination rates, one or two years of IPV administration after OPV cessation could be sufficient to prevent outbreaks of paralytic poliomyelitis from vaccine-derived strains.
Polio vaccine suspected as cause of fatal mutant form of encephalitis
The polio vaccine isn’t protecting children – and, worse, it appears to be causing a new and sometimes fatal form of the disease.
Concerns about the vaccine have arisen following a high number of deaths and hospital admissions from encephalitis and polio in the Uttar Pradesh region of India – where there has been an intensive vaccination programme.
Around 400 children have died, and a further 2,300 admitted to hospital, following an outbreak of a new form of viral encephalitis, and doctors admit they do not know its cause.
Unvaccinated Blamed for Mutated Polio, (AGAIN ALWAYS FALSELY THE UNVACCINATED ARE BLAMED FOR ANYTHING THAT HAPPENS)
Mutant polio vaccine regains virulence
But the latest study raises the frightening possibility that the vaccine strain can also regain the ability to spread between people more easily than thought. "It demonstrates clearly that the vaccine virus can spread from person to person," says Olen Kew from the Centers for Disease Control and Prevention in Atlanta, Georgia.
The outbreak was exacerbated by the fact that Haiti had relaxed its polio vaccination program more than five years earlier. "It's a warning that you need to have good coverage to prevent vaccines from running away like this," Kew says.
The study also shows how difficult it will be eliminate polio entirely. For this to be achieved, natural polio would first have to be wiped out through stringent use of the oral polio vaccine. Then all countries could simultaneously stop vaccinating or switch to a different vaccine - injectable, dead polio virus.
This method does not confer as much immunity as the oral vaccine, but it cannot revert to a disease-causing form. This vaccine is already used in the US and much of Europe.
This again points to the claim that they think they need to get 100% vaccine coverage in ever country with existing polio, and only then it may be possible to stop polio, but yet they know they will have the mutations still going on and the result of that is in their minds quite obviously only necessary collateral damage, so to speak. So, as long as they can keep blaming it all on the unvaccinated, which is not exactly proven; it is an assumption. And as long as they keep playing Russian Roulette with the vaccine virus; in the hopes that it does not continue to mutate to a point of becoming a super virus world wide. But in the end, the with the known odds that have been and in the resulting outcomes; clearly it all shows this plan to be not only failing and dangerous; but even currently, is likely causing more harm than good; and will continue to.
J Clin Microbiol. 1995 Sep;33(9):2485-8.
Detection of measles virus RNA in urine specimens from vaccine recipients.
Source: Division of Viral and Rickettsial Diseases, Centers for Disease Control and Prevention, Atlanta, Georgia 30333, USA.
Analysis of urine specimens by using reverse transcriptase-PCR was evaluated as a rapid assay to identify individuals infected with measles virus. For the study, daily urine samples were obtained from either 15-month-old children or young adults following measles immunization. Overall, measles virus RNA was detected in 10 of 12 children during the 2-week sampling period. In some cases, measles virus RNA was detected as early as 1 day or as late as 14 days after vaccination. Measles virus RNA was also detected in the urine samples from all four of the young adults between 1 and 13 days after vaccination. This assay will enable continued studies of the shedding and transmission of measles virus and, it is hoped, will provide a rapid means to identify measles infection, especially in mild or asymptomatic cases.
You see in the next below link that it ALL depends on who has done the study, as for if they find the evidence of shedding due to a/or the vaccine. Here we have the Journal of Infectious diseases that is closely aligned with pharma and Offit's CHOP. And they of course find predicable no shedding. Can you imagine the upset if they had, and presented to the CDC with that? Clearly, is not happening.
J Infect Dis. 2004 May 1;189 Suppl 1:S165-70.
Lack of evidence of measles virus shedding in people with inapparent measles virus infections.
So, the pro vaccine side again claims to what? Well if there are no studies to prove that the vaccines cause shedding, then it simply doesn't happen. Just like in regard to the vaccine aluminum adjuvants; if no studies have ever been done, then we can proclaim that there is no scientific proof of the harm, thus there is no said harm being done.
J Clin Microbiol. 2008 Mar;46(3):1101-3. Epub 2008 Jan 9.
Detection of RNA of mumps virus during an outbreak in a population with a high level of measles, mumps, and rubella vaccine coverage.
Source:Epidemic Intelligence Service, Office of Workforce and Career Development, Centers for Disease Control and Prevention, Atlanta, GA 30333, USA.
The duration of mumps virus RNA detection was studied during a mumps outbreak in a highly vaccinated university population. Seven of the eight reverse transcription-PCR-positive specimens were collected during the first 3 days of parotitis, suggesting that viral shedding is minimal after the first 3 days of symptoms.